Professor Ed Sturrock

Zinc Metalloprotease Research Group

Affiliations

  1. Full Member, Institute of Infectious Disease and Molecular Medicine
  2. Senior Research Scholar and Emeritus Professor, Division of Chemical and Systems Biology, Department of Integrative Biomedical Sciences, University of Cape Town (UCT)
  3. Fellow of Royal Society of South Africa; Fellow of UCT; Member of ASSAf

Key Expertise

Protein biochemistry

Main Research Focus

Structure-function aspects of angiotensin-converting enzyme (ACE); design and synthesis of novel domain-selective ACE and vasopeptidase inhibitors for hypertension and CVD; the mechanisms involved in fibrosing tuberculous pericarditis; and the processing of the membrane-anchored proteins. Ed has published over 120 peer-reviewed papers and five patents, and has trained more than 40 students at PhD and MSc levels. He currently holds an NRF A rating. Together with colleagues in the Unites States (US) and United Kingdom (UK), he founded a spin-off company AngioDesign (UK) Ltd.

Most Significant Paper Authored in 2024

Proteomic analysis of human macrophages 1 overexpressing angiotensin converting enzyme.

Oosthuizen D, Ganief TA, Bernstein KE, Sturrock ED (2024).


The success of an angiotensin converting enzyme (ACE)-overexpressing murine macrophage model in treating microbial infections and cancer opens a new avenue into whether ACE overexpression in human macrophages shares these benefits. In the present study, proteomic changes in an ACE-overexpressing THP-1 cell line were assessed using mass spectrometry. ACE activity was significantly reduced following inhibitor treatment, and both RNA processing and immune pathways were significantly dysregulated. A novel, functionally enriched immune pathway that appeared both with ACE overexpression and inhibitor treatment was neutrophil degranulation. ACE overexpression within human macrophages showed similarities with ACE 10/10 murine macrophages, paving the way for mechanistic studies aimed at understanding the altered immune function.